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In conclusion, we found that the stress of HT on cucumber seedlings was mostly relieved by the exogenous supplementation of GSH

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They reported that CHE inhibited cell proliferation by triggering mitochondrial apoptosis through the PI3K/BAD signaling pathway, significantly increasing the activated expression of the BAD protein, upregulating the proapoptotic proteins BAX and BAK, and downregulating the antiapoptotic proteins BCL-2 and MCL-1 (Yang et al., 2020)

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