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Epidermal growth factor receptor (EGFR) activation was identified as a basis for the lower response in CRC, with additional EGFR inhibition together with BRAF inhibitor treatment overcoming this issue [7]

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Key mechanisms include estrogen dominance with progesterone resistance, immune dysfunction, oxidative stress-induced ferroptosis, and disrupted endometrial receptivity

Oxidative stress by ascorbate/menadione association kills K562 human chronic myelogenous leukaemia cells and inhibits its tumour growth in nude mice