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The same observations were found in rat brains in vivo (Morello et al., 2007) and in dopamine neurons (Neely et al., 2017) and SH-SY5Y cells in vitro (Maddirala et al., 2015)
Instead of being filtered and excreted by kidney, those uremic toxins accumulate in blood and lead to a deficient erythropoietin (EPO) production by the kidney interstitial fibroblasts (peritubular cells of kidneys), resulting in a defective erythropoiesis due to a decrease in erythrocyte production [9,10]
Redox regulation of RAD51 Cys319 and homologous recombination by peroxiredoxin 1
